Public Health Impacts
Superfund Research Program
One of the primary goals of SRP-funded research is to improve public health. Thus, the Program supports a wide range of research to address the broad public health concerns arising from the release of hazardous substances into the environment. The intent is to provide sound science to those making public policy, regulatory, and risk reduction decisions. SRP-funded research has been successful in this area as studies have improved our understanding of the health effects associated with exposures to environmental contaminants. The following stories provide information on public health impacts. They are merely highlights and represent the breadth of work SRP researchers undertake. To see older stories, visit our archives page.
A new technology, developed with NIEHS funding, will provide safe drinking water to California communities at approximately half the cost of other options and with virtually no secondary waste. NIEHS Superfund Research Program (SRP) small business grantee Microvi Biotechnologies celebrated installation of its advanced nitrate removal technology during a grand opening Jan. 25 at Sunny Slope Water Company. The company delivers water to 30,000 households in southern California, and the new system will provide more than 200 million gallons of treated water to its customers.
The U.S. Food and Drug Administration (FDA) issued a rule banning 19 antibacterial chemicals as ingredients in over-the-counter (OTC) antibacterial hand and body washes. Development of the final rule was informed by research that included several studies from scientists supported by the NIEHS Superfund Research Program (SRP). The final rule bans the sale of any OTC consumer soaps and body washes containing any of the banned chemicals, including triclosan and triclocarban, unless the product undergoes approval as a drug.
Britton Goodale, Ph.D., is a postdoctoral fellow at Dartmouth College under the guidance of Bruce Stanton, Ph.D. Goodale's SRP research focuses on how low-dose arsenic exposure affects the ability of human airway cells to respond to bacterial infections.