Inside the Institute
Calendar of Upcoming Events
May 01, Keystone 3003 12:00 p.m.–1:00 p.m. — “Prenatal Exposure to Persistent Pollutants in a 20 Year Old Danish Cohort: Results and Future Direction,” by Thorhallur Halldorsson, Ph.D.
May 02, Rall F-193 11:00 a.m.–12:00 p.m. — Laboratory of Toxicology and Pharmacology hosted presentation on “Autism Spectrum Disorders and Shank3 Disruption in Mice,” by William Wetsel, Ph.D.
May 03, Rodbell A 11:00 a.m.–12:00 p.m. — Laboratory of Reproductive and Developmental Toxicology hosted seminar on “Phenobarbital Indirectly Activates the Constitutive Active Androstane Receptor (CAR) by Inhibition of Epidermal Growth Factor Receptor Signaling and the Function of Transactivation Domains of Estrogen Receptor A,” by Shingo Mutoh, Ph.D., and Yukitomo Arao, Ph.D.
May 08, Rodbell Auditorium 11:00 a.m.–12:00 p.m. — Distinguished Lecture Series featuring
Marie Davidian, Ph.D., discussing “The Right Treatment for the Right Patient (at the Right Time)”
May 14, Rodbell Auditorium 8:30 a.m.–5:00 p.m. — National Advisory Environmental Health Sciences Council
May 21, Rall F-193 12:30 p.m.–2:00 p.m. — DNA Repair Videoconference: Human DNA Repair Disorders, a Historical Perspective,” by Alessandro Vindigni, Ph.D.
May 21 (Offsite event), Room C111A, at U.S. Environmental Protection Agency in Research Triangle Park, N.C. 8:00 a.m.–4:00 p.m. — Genetics and Environmental Mutagenesis Society (GEMS) Spring Symposium, “Mechanisms of Environmental Carcinogenesis”
View More Events: NIEHS Public Calendar
Extramural papers of the month/news/newsletter/2013/5/dert/index.htm
- Prenatal DDT exposure associated with high blood pressure in adults
- Blocking glucocorticoid receptor prevents arsenic-induced birth defects
- Community beliefs regarding dioxin exposure pathways
- BPA exposure in the NICU
Intramural papers of the month/news/newsletter/2013/5/dir/index.htm
- A novel mechanism underlies glucocorticoid resistance
- COX-2 involved in the development and worsening of asthma symptoms
- Early life socioeconomic factors influence the development of rheumatoid arthritis
- Specific DNA polymerase active site residue may influence a cell’s mutagenic response to oxidative stress